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Month: February 2019

In order to remain healthy and functional, cells have a number of maintenance systems that help them to dispose of metabolic waste and unwanted proteins. Autophagy is perhaps the best-known example of how cells purge their waste, and another is the ubiquitin-proteasome system (UPS). Researchers are working on ways to boost the activity of the...
On Monday, April 8th, we will be hosting our second Lifespan.io webinar, during which we will be discussing the microbiome and its role in aging and disease. Our second research webinar Our work is largely supported by the generosity of our monthly patrons, the Lifespan Heroes, so to thank them we have launched a new...
EARD2019 conference banner.
July 2019 will see the launch of our second Ending Age-Related Diseases conference at the Frederick P. Rose Auditorium, Cooper Union in New York City. The event was so popular last year that we decided to expand it to two full days of science and biotech business this year. We will be bringing you the...
In a new study [1], researchers have identified the reason why cells become defective when they grow too large and why protein creation fails when cells grow larger than their original healthy size, as is typically seen in aged and senescent cells. They demonstrate that in enlarged yeast and human cells, RNA and protein biosynthesis...
Researchers at the Harvard T.H. Chan School of Public Health have discovered a new aging clock that can accurately determine both chronological and biological age in a wide variety of species. Aging and the nucleolus There are two kinds of age: chronological age, which is strictly the number of years that something has lived, and...
I recently visited the Longevity Leaders Conference in London and had the opportunity to speak with Kelsey Moody, the CEO of Ichor Therapeutics, a company focused on targeting age-related diseases by targeting the aging processes themselves. I previously interviewed him back in 2017, so it was the ideal time to catch up on what had...